Known … DOI: https://doi.org/10.1523/JNEUROSCI.21-12-04195.2001 Ultrastructure of the synapses of sensory neurons that mediate the gill-withdrawal reflex in Involvement of pre- and postsynaptic mechanisms in post-tetanic potentiation at Computer modeling of synapsin I binding to synaptic vesicles and F-actin: implications for the regulation of neurotransmitter release.Interaction of free and synaptic vesicle-bound synapsin I with F-actin.Interactions of synapsin I with phospholipids: possible role in synaptic vesicle clustering and in the maintenance of bilayer structures.Protein-protein interactions and protein modules in the control of neurotransmitter release.Cycling of actin assembly in synaptosomes and neurotransmitter release.Sustained neurotransmitter release: new molecular clues.Dephosphorylated synapsin I anchors synaptic vesicles to actin cytoskeleton: an analysis by videomicroscopy.Heterogeneity of release probability, facilitation, and depletion at central synapses.Calcium-dependent regulation of Rab3 in short-term plasticity.A role for synaptotagmin (p65) in regulated exocytosis.Synapsin I is structurally similar to ATP-utilizing enzymes.Bilateral symmetry and interneuronal organization in the buccal ganglia of Improving the quality of immunoblots by chromatography of polyclonal antisera on keratin affinity columns.Synaptic vesicle phosphoproteins and regulation of synaptic function.Two sites of action for synapsin domain E in regulating neurotransmitter release.The cytoskeletal architecture of the presynaptic terminal and molecular structure of synapsin I.Synapsin III, a novel synapsin with an unusual regulation by CaA phospho-switch controls the dynamic association of synapsins with synaptic vesicles.Evidence for a functional link between Rab3 and the SNARE complex.Invertebrate synapsins: a single gene codes for several isoforms in Kinetics and regulation of fast endocytosis at hippocampal synapses.Post-tetanic potentiation at an identified synapse in Two distinct pools of synaptic vesicles in single presynaptic boutons in a temperature-sensitive Cleavage of structural proteins during the assembly of the head of bacteriophage T4.The organization of cytoplasm at the presynaptic active zone of a central nervous system synapse.Impairment of synaptic vesicle clustering and of synaptic transmission and increased seizure propensity, in synapsin I-deficient mice.Exogenous synapsin I promotes functional maturation of developing neuromuscular synapses.An analysis of facilitation of transmitter release at the neuromuscular junction of the frog.Opposite actions of nitric oxide on cholinergic synapses: which pathways?Reversal of synaptic vesicle docking at central synapses.Phosphorylation-dependent effects of synapsin IIa on actin polymerization and network formation.Distinct pools of synaptic vesicles in neurotransmitter release.Antibodies against rat brain vesicle-associated membrane protein (synaptobrevin) prevent inhibition of acetylcholine release by tetanus toxin or botulinum neurotoxin type B.Short-term synaptic plasticity is altered in mice lacking synapsin I.Essential functions of synapsin I and II in synaptic vesicles regulation.Definition of the readily releasable pool of vesicles at hippocampal synapses.The kinetics of synaptic vesicle recycling measured at single presynaptic boutons.Synaptic vesicle recycling in synapsin I knock-out mice.Tetanus and botulinum-B neurotoxins block neurotransmitter release by proteolytic cleavage of synaptobrevin.Characterization of synapsin I binding to small synaptic vesicles.Synapsin IIa: expression in insect cells, purification and characterization.Estimates for the pool size of releasable quanta at a single central synapse and for the time required to refill the pool.Synapsins: mosaics of shared and individual domains in a family of synaptic vesicle phosphoproteins.Role of residual calcium in synaptic depression and posttetanic potentiation: fast and slow calcium signaling in nerve terminals.Synapsin I deficiency results in structural changes in the presynaptic terminals in the murine nervous system.VAMP (synaptobrevin) is present in the plasma membrane of nerve terminals.Impairment of inhibitory synaptic transmission in mice lacking synapsin I.Synapsin I partially dissociates from synaptic vesicles during exocytosis induced by electrical stimulation.Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedures and some applications.Accelerated structural maturation induced by synapsin I at developing neuromuscular synapses of The t-SNAREs syntaxin 1 and SNAP-25 are present on organelles that participate in synaptic vesicle recycling.Exocytosis: a molecular and physiological perspective.Calcium- and activity-dependent synaptic plasticity.Evidence for Long-Lasting Cholinergic Control of Gap Junctional Communication between Adrenal Chromaffin CellsMenstrual Cycle-Dependent Neural Plasticity in the Adult Human Brain Is Hormone, Task, and Region SpecificCalcium Influx via L- and N-Type Calcium Channels Activates a Transient Large-Conductance CaDevelopmentally Transient CB1Rs on Cerebellar Afferents Suppress Afferent Input, Downstream Synaptic Excitation, and Signaling to Migrating NeuronsDeletion of the Mitochondrial Matrix Protein CyclophilinD Prevents Parvalbumin Interneuron Dysfunctionand Cognitive Deficits in a Mouse Model of NMDA Hypofunction
We do not retain these email addresses.Fiumara F, Ghirardi M, Onofri F, Benfenati F, Montarolo PG (2001) Intracellular injection of synapsin I induces neurotransmitter release in C1 neurons of Enter multiple addresses on separate lines or separate them with commas.This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.Synapsin Controls Both Reserve and Releasable Synaptic Vesicle Pools during Neuronal Activity and Short-Term Plasticity inSynapsin Controls Both Reserve and Releasable Synaptic Vesicle Pools during Neuronal Activity and Short-Term Plasticity in Anne Humeau, Benjamin Buard, David Rousseau, Francois Chapeau-Blondeau, Pierre Abraham, Multifractal Analysis of Laser Doppler Flowmetry Signals: Partition Function and Generalized Dimensions of Data Recorded before and after Local Heating, Biocybernetics and Biomedical Engineering, 10.1016/S0208-5216(12)70029-3, 32, 1, (17-26), (2012). Search by Name Search Tools. Name. A major regulation concerns the maintenance of a reserve pool that confers the nerve terminal the ability to release neurotransmitter at firing rates exceeding the maximum SV recycling capacity and to dynamically change the efficiency of release in response to conditioning stimuli (e.g., the PTP) by transiently shifting the equilibrium between the reserve and the releasable SV pools (Fig. Acoustique ~ Release by Bernard Lavilliers (see all versions of this release, 3 available) In addition, nerve terminals are able to transiently modify synaptic efficacy to give rise to various forms of short-term plasticity. Available for sale from Michele Mariaud Gallery, Pascal Pierme, Petite Tatoum (2018), Steel, 21 × 13 × 7 in Pascal Bray. Thomas Humeau. The financial support of Telethon–Italy (Grant 1131 to F.B.) As detailed below, depending on the stimulation rate, the underlying synapsin-dependent mechanisms altered by antibody injection seem to be different.An experimental situation to be considered is the depression induced by sustained 0.5 Hz stimulation (Fig. Although the vertebrate synapsin family comprises the products of at least three synapsin genes (synapsin I, II, and III) (In this paper, we took advantage of well characterized cholinergic synapses in the buccal ganglion of The ability of the antibodies directed against mammalian synapsins to recognize the Effects of anti-synapsin antibody injection on ACh release. Phone: +41 (0) 22 748 18 29. Copyright 2020 PeekYou.com. Regarding the mechanisms of synapsin “neutralization” by the antibodies, it seems unlikely that the antibodies are able to enter the thick clusters of vesicles and actin filaments and directly compete with the binding of synapsin to vesicles and actin.
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